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References from Blockade of the Fas-Fas Ligand Pathway Inhibits Cyclophosphamide-induced Diabetes in NOD Mice. Local targets link to admitted publications; unresolved targets remain external evidence.
Type-I diabetes: a chronic autoimmune disease of human, mouse, and rat
10.1146/annurev.iy.08.040190.003243 · 1990 · External reference
Insulin-dependent diabetes mellitus as an autoimmune disease
10.1210/edrv-15-4-516 · 1994 · External reference
The nonobese diabetic mouse as a model of autoimmune diabetes: immune dysregulation gets the NOD
10.1016/s1074-7613(00)80392-1 · 1997 · External reference
Apoptosis is the mode of beta-cell death responsible for the development of IDDM in the nonobese diabetic (NOD) mouse
10.2337/diab.46.5.750 · 1997 · External reference
Beta cell apoptosis in T cell-mediated autoimmune diabetes
10.1073/pnas.94.1.213 · 1997 · External reference
The role of Fas in autoimmune diabetes
10.1016/s0092-8674(00)80178-6 · 1997 · External reference
Requirement of Fas for the development of autoimmune diabetes in nonobese diabetic mice
10.1084/jem.186.4.613 · 1997 · External reference
Inhibition of autoimmune diabetes by Fas ligand: the paradox is solved
10.4049/jimmunol.164.6.2931 · 2000 · External reference
Significant role for Fas in the pathogenesis of autoimmune diabetes
10.4049/jimmunol.164.5.2523 · 2000 · External reference
10.1073/pnas.95.23.13818
10.1073/pnas.95.23.13818 · External reference
In autoimmune diabetes the transition from benign to pernicious insulitis requires an islet cell response to tumor necrosis factor alpha
10.1084/jem.189.7.1053 · 1999 · External reference
Apoptosis of pancreatic beta-cells detected in accelerated diabetes of NOD mice: no role of Fas-Fas ligand interaction in autoimmune diabetes
10.1002/(sici)1521-4141(199902)29:02<455::aid-immu455>3.0.co;2-a · 1999 · External reference
Beta-cell destruction in NOD mice correlates with Fas (CD95) expression on beta-cells and proinflammatory cytokine expression in islets
10.2337/diabetes.48.1.21 · 1999 · External reference
Promotion of spontaneous diabetes in non-obese diabetes-prone mice by cyclophosphamide
10.1007/bf00276978 · 1984 · External reference
Cyclophosphamide-induced diabetes in NOD/WEHImice. Evidence for suppression in spontaneous autoimmune diabetes mellitus
10.2337/diab.38.4.441 · 1989 · External reference
Unresolved reference
External reference
Anti-suppressor effect of cyclophosphamide on the development of spontaneous diabetes in NOD mice
10.1002/eji.1830180325 · 1988 · External reference
Programmed cell death in the pathogenesis of murine IDDM: Resistance to apoptosis induced in lymphocytes by cyclophosphamide
10.1006/jaut.1996.0034 · 1996 · External reference
Augmentation of delayed-type hypersensitivity by doses of cyclophosphamide which do not affect antibody responses
10.1084/jem.141.3.697 · 1975 · External reference
Regulation of delayed-type hypersensitivity reactions by cyclophosphamide-sensitive T cells
10.4049/jimmunol.121.4.1573 · 1978 · External reference
Reinduction of experimental allergic encephalomyelitis in convalescent Lewis rats with cyclophosphamide
10.1016/0022-510x(85)90152-2 · 1985 · External reference
Experimental allergic encephalomyelitis in the Lewis rat. A model of predictable relapse by cyclophosphamide
10.1016/0022-510x(87)90063-3 · 1987 · External reference
Early quantitative and functional deficiency of NK1(+)- like thymocytes in the NOD mouse
10.1002/eji.1830261226 · 1996 · External reference
Lack of L-selectin expression by cells transferring diabetes in NOD mice: insights into the mechanisms involved in diabetes prevention by Mel-14 antibody treatment
10.1002/eji.1830250605 · 1995 · External reference
Characterization of peripheral regulatory CD4(+) T cells that prevent diabetes onset in nonobese diabetic mice
10.4049/jimmunol.164.1.240 · 2000 · External reference
Mature mainstream TCR alpha beta+CD4+ thymocytes expressing L-selectin mediate ‘active tolerance’ in the nonobese diabetic mouse
10.4049/jimmunol.161.5.2620 · 1998 · External reference
T cell-mediated inhibition of the transfer of autoimmune diabetes in NOD mice
10.1084/jem.169.5.1669 · 1989 · External reference
Fas involvement in Ca(2+)-independent T cell-mediated cytotoxicity
10.1084/jem.177.1.195 · 1993 · External reference
The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis
10.1016/0092-8674(91)90614-5 · 1991 · External reference
Monoclonal antibody-mediated tumor regression by induction of apoptosis
10.1126/science.2787530 · 1989 · External reference
The TNF receptor superfamily of cellular and viral proteins: activation, costimulation, and death. [Review] [26 refs]
10.1016/0092-8674(94)90372-7 · 1994 · External reference
The role of APO-1-mediated apoptosis in the immune system. [Review]
10.1111/j.1600-065x.1994.tb00889.x · 1994 · External reference
The Fas death factor
10.1126/science.7533326 · 1995 · External reference
The fas antigen is involved in peripheral but not thymic deletion of T lymphocytes in T cell receptor transgenic mice
10.1016/1074-7613(94)90067-1 · 1994 · External reference
CD95 (Fas)-dependent elimination of self-reactive B cells upon interaction with CD4+ T cells
10.1038/376181a0 · 1995 · External reference
Molecular cloning and expression of the Fas ligand, a novel member of the tumor necrosis factor family
10.1016/0092-8674(93)90326-l · 1993 · External reference
Expression and function of mouse Fas antigen on immature and mature T cells
10.4049/jimmunol.154.9.4395 · 1995 · External reference
Activated B cells express functional Fas ligand
10.1002/eji.1830260332 · 1996 · External reference
A role for CD95 ligand in preventing graft rejection
10.1038/377630a0 · 1995 · External reference
Fas ligand-induced apoptosis as a mechanism of immune privilege
10.1126/science.270.5239.1189 · 1995 · External reference
Prevention of islet allograft rejection with engineered myoblasts expressing FasL in mice
10.1126/science.273.5271.109 · 1996 · External reference
Melanoma cell expression of Fas(Apo-1/CD95) ligand: implications for tumor immune escape
10.1126/science.274.5291.1363 · 1996 · External reference
Fas-and FasL-deficient mice are resistant to induction of autoimmune encephalomyelitis
10.4049/jimmunol.159.7.3100 · 1997 · External reference
Potential involvement of Fas and its ligand in the pathogenesis of Hashimoto's thyroiditis [see comments]
10.1126/science.275.5302.960 · 1997 · External reference
The Fas/Fas ligand system is involved in the pathogenesis of autoimmune myocarditis in rats
10.4049/jimmunol.161.9.4695 · 1998 · External reference
Cyclophosphamide therapy of severe lupus nephritis
10.1016/s0272-6386(97)90097-4 · 1997 · External reference
Cyclophosphamide therapy in systemic lupus erythematosus. [Review in German]
10.1007/s003930050033 · 1997 · External reference
Alkylating agents
1985 · External reference
The effect of cyclophosphamide treatment on lymphocyte subsets in the nonobese diabetic mouse: a comparison of various lymphoid organs
10.3109/08916939309004833 · 1993 · External reference
Protection against Fas-dependent Th1-mediated apoptosis by antigen receptor engagement in B cells
10.1038/374163a0 · 1995 · External reference
Expression of Fas in B cells of the mouse germinal center and Fas-dependent killing of activated B cells
10.1093/intimm/7.12.1949 · 1995 · External reference
B cells in systemic autoimmune disease: recent insights from Fas-deficient mice and men
10.1016/s0952-7915(96)80015-x · 1996 · External reference
Drug-induced apoptosis in hepatoma cells is mediated by the CD95 (APO-1/Fas) receptor/ligand system and involves activation of wild-type p53
10.1172/jci119174 · 1997 · External reference
Involvement of the CD95 (APO-1/FAS) receptor/ligand system in drug-induced apoptosis in leukemia cells
10.1038/nm0596-574 · 1996 · External reference
B7/CD28 Costimulation is essential for the homeostasis of the CD4+CD25+ immunoregulatory T cells that control autoimmune diabetes
10.1016/s1074-7613(00)80195-8 · 2000 · External reference
Down-regulation of homing receptors after T cell activation
10.4049/jimmunol.141.12.4110 · 1988 · External reference
Differential ability of Th1 and Th2 T cells to express Fas ligand and to undergo activation-induced cell death
10.1093/intimm/6.10.1545 · 1994 · External reference
Syngeneic transfer of autoimmune diabetes from diabetic NOD mice to healthy neonates. Requirement for both L3T4+ and Lyt-2+ T cells
10.1084/jem.166.4.823 · 1987 · External reference
Both the Lyt-2+ and L3T4+ T cell subsets are required for the transfer of diabetes in nonobese diabetic mice
10.4049/jimmunol.140.1.52 · 1988 · External reference
Analysis of the roles of CD4+ and CD8+ T cells in autoimmune diabetes of NOD mice using transfer to NOD athymic nude mice
10.1002/eji.1830220931 · 1992 · External reference
Pancreatic islet-specific T-cell clones from nonobese diabetic mice
10.1073/pnas.86.20.8000 · 1989 · External reference
T helper cell subsets in insulin-dependent diabetes
10.1126/science.7761837 · 1995 · External reference
In vivo activity and in vitro specificity of CD4+ Th1 and Th2 cells derived from the spleens of diabetic NOD mice
10.1172/jci118006 · 1995 · External reference
