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Conversion of zonulae occludentes from tight to leaky strand type by introducing claudin-2 into Madin–Darby canine kidney I cells

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Are alterations of tight junctions at molecular and ultrastructural level different in duodenal biopsies of patients with celiac disease and Crohn׳s disease?

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Claudins and the modulation of tight junction permeability

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Interleukin-13 is the key effector Th2 cytokine in ulcerative colitis that affects epithelial tight junctions, apoptosis, and cell restitution

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Diarrhea-predominant irritable bowl syndrome: an organic disorder with structural abnormalities in the jejunal epithelial barrier

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Claudin-3 acts as a sealing component of the tight junction for ions of either charge and uncharged solutes

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Autophagy enhancement of intestinal epithelial tight junction barrierfunction by targeting claudin-2 degradation

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Changes in the expression of claudins in active ulcerative colitis

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Inflammatory processes have differential effects on claudins 2, 3 and 4 in colonic epithelial cells

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Claudin-2, a component of the tight junction, forms a paracellular water channel

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Altered tight junction structure contributes to the impaired epithelial barrier function in ulcerative colitis

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Cell polarity-determining proteins Par-3 and PP-1 are involved in epithelial tight junction defects in coeliac disease

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Tight junction pore and leak pathways: a dynamic duo

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Regulation of intestinal epithelial permeability by tight junctions

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Mucosal expression of claudins 2, 3 and 4 in proximal and distal part of duodenum in children with coeliac disease

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Interleukin-6 (IL-6) regulates claudin-2 expression and tight junction permeability in intestinal epithelium

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Loss of claudin-15, but not claudin-2, causes Na+ deficiency and glucose malabsorption in mouse small intestine

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Urine-based detection of intestinal tight junction loss

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Loss of claudins 2 and 15 from mice causes defects in paracellular Na+ flow and nutrient transport in gut and leads to death from malnutrition

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Claudin-1 and claudin-2 expression is elevated in inflammatory bowel disease and may contribute to early neoplastic transformation

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Changes in expression and distribution of claudin 2, 5 and 8 lead to discontinuous tight junctions and barrier dysfunction in active Crohn׳s disease

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