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References from Bax and caspases regulate increased production of mitochondria-derived reactive species in neuronal apoptosis: LACK of A role for depletion of cytochrome c from the mitochondrial electron transport chain. Local targets link to admitted publications; unresolved targets remain external evidence.
Cell death during development of the nervous system
10.1146/annurev.ne.14.030191.002321 · 1991 · External reference
Inhibitors of protein synthesis and RNA synthesis prevent neuronal death caused by nerve growth factor deprivation
10.1083/jcb.106.3.829 · 1988 · External reference
Caspase inhibition extends the commitment to neuronal death beyond cytochrome c release to the point of mitochondrial depolarization
10.1083/jcb.150.1.131 · 2000 · External reference
Inhibition of apoptotic signaling cascades causes loss of trophic factor dependence during neuronal maturation
10.1083/jcb.149.5.1011 · 2000 · External reference
Bax Regulates Production of superoxide in both apoptotic and nonapoptotic neurons: role of caspases
10.1523/jneurosci.2862-10.2010 · 2010 · External reference
Bax is required for neuronal death after trophic factor deprivation and during development
10.1016/s0896-6273(00)80173-7 · 1996 · External reference
Intrinsic and extrinsic pathway signaling during neuronal apoptosis: lessons from the analysis of mutant mice
2002 · External reference
Movement of Bax from the cytosol to mitochondria during apoptosis
10.1083/jcb.139.5.1281 · 1997 · External reference
Bax translocation is a critical event in neuronal apoptosis: regulation by neuroprotectants, Bcl-2, and caspases
10.1523/jneurosci.19-17-07476.1999 · 1999 · External reference
Rapid activation of antioxidant defenses by nerve growth factor suppresses reactive oxygen species during neuronal apoptosis: evidence for a role in cytochrome c redistribution
10.1523/jneurosci.3590-07.2007 · 2007 · External reference
Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade
10.1016/s0092-8674(00)80434-1 · 1997 · External reference
Cell Death: Critical Control Points
10.1016/s0092-8674(04)00046-7 · 2004 · External reference
Mitochondria and cell death: outer membrane permeabilization and beyond
10.1038/nrm2952 · 2010 · External reference
Superoxide dismutase delays neuronal apoptosis: a role for reactive oxygen species in programmed neuronal death
10.1016/0896-6273(95)90287-2 · 1995 · External reference
Rapid suppression of free radical formation by nerve growth factor involves the mitogen-activated protein kinase pathway
10.1073/pnas.94.8.4086 · 1997 · External reference
Evidence for redox regulation of cytochrome c release during programmed neuronal death: antioxidant effects of protein synthesis and caspase inhibition
10.1523/jneurosci.21-06-01949.2001 · 2001 · External reference
A Bax-induced pro-oxidant state is critical for cytochrome c release during programmed neuronal death
10.1523/jneurosci.22-15-06480.2002 · 2002 · External reference
Disruption of mitochondrial function during apoptosis is mediated by caspase cleavage of the p75 subunit of complex I of the electron transport chain
10.1016/j.cell.2004.05.008 · 2004 · External reference
Caspase-mediated loss of mitochondrial function and generation of reactive oxygen species during apoptosis
10.1083/jcb.200208089 · 2003 · External reference
Superoxide in apoptosis: mitochondrial generation triggered by cytochrome c loss
10.1074/jbc.273.19.11401 · 1998 · External reference
Regulation of hydrogen peroxide production by brain mitochondria by calcium and Bax
10.1046/j.1471-4159.2002.01153.x · 2002 · External reference
, Bid-induced mitochondrial membrane permeabilization waves propagated by local reactive oxygen species (ROS) signaling
2011 · External reference
Bax-deficient mice with lymphoid hyperplasia and male germ cell death
10.1126/science.270.5233.96 · 1995 · External reference
Evaluation of 2′, 7′-dichlorofluorescin and dihydrorhodamine123 as fluorescent probes for intracellular H2O2 in cultured endothelial cells
10.1006/abbi.1993.1222 · 1993 · External reference
Mitochondria and neuronal survival
10.1152/physrev.2000.80.1.315 · 2000 · External reference
Mitochondrial membrane potential and cell death: mortality and millivolts
10.1016/s0166-2236(99)01534-9 · 2000 · External reference
Unresolved reference
2002 · External reference
Unresolved reference
2007 · External reference
The regulation of reactive oxygen species production during programmed cell death
10.1083/jcb.141.6.1423 · 1998 · External reference
Widespread elimination of naturally occurring neuronal death in Bax-deficient mice
10.1523/jneurosci.18-04-01428.1998 · 1998 · External reference
Cytochrome c is released in a single step during apoptosis
10.1038/sj.cdd.4401596 · 2005 · External reference
Inhibition of NGF deprivation-induced death by low oxygen involves suppression of BIMEL and activation of HIF-1
10.1083/jcb.200407079 · 2005 · External reference
Alternating metabolic pathways in NGF deprived sympathetic neurons affect caspase-independent death
10.1083/jcb.200302109 · 2003 · External reference
Cytochrome c release from mitochondria proceeds by a two-step process
10.1073/pnas.241655498 · 2002 · External reference
Mitochondrial membrane potential regulates matrix configuration and cytochrome c release during apoptosis
10.1038/sj.cdd.4401231 · 2003 · External reference
The role of the Bcl-2 family in the regulation of outer mitochondrial membrane permeability
10.1038/sj.cdd.4400781 · 2000 · External reference
Bcl-2 functions in an antioxidant pathway to prevent apoptosis
10.1016/0092-8674(93)80066-n · 1993 · External reference
Shift of the cellular oxidation-reduction potential in neural cells expressing Bcl-2
10.1046/j.1471-4159.1996.67031259.x · 1996 · External reference
Bcl-2 family proteins regulate mitochondrial reactive oxygen production and protect against oxidative stress
10.1016/j.freeradbiomed.2004.09.005 · 2004 · External reference
