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References from The Progress of CRISPR/Cas9-Mediated Gene Editing in Generating Mouse/Zebrafish Models of Human Skeletal Diseases. Local targets link to admitted publications; unresolved targets remain external evidence.
Continuous remodeling of the skeleton. Growth factors and cytokines direct the activity
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Unresolved reference
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Optimized knock-in of point mutations in zebrafish using CRISPR/Cas9
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PFKFB4 control of AKT signaling is essential for premigratory and migratory neural crest formation
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