Research graph
References from IL-13-Amphiregulin pathway exacerbates itch in allergic contact dermatitis. Local targets link to admitted publications; unresolved targets remain external evidence.
Misregulation of T-box transcription factors drives BNP-NPR1 signalling underlying chronic itch
10.1038/s41467-026-75726-x · 2026 · External reference
Novel insights into the TRPV3-mediated itch in atopic dermatitis
10.1016/j.jaci.2020.09.028 · 2021 · External reference
Defining cell type-specific immune responses in a mouse model of allergic contact dermatitis by single-cell transcriptomics
10.7554/elife.94698.3 · 2024 · External reference
Amphiregulin-Producing Pathogenic Memory T Helper 2 Cells Instruct Eosinophils to Secrete Osteopontin and Facilitate Airway Fibrosis
10.1016/j.immuni.2018.04.023 · 2018 · External reference
Enhanced expression levels of IL-31 correlate with IL-4 and IL-13 in atopic and allergic contact dermatitis
10.1016/j.jaci.2006.07.015 · 2006 · External reference
Single-cell transcriptomics combined with interstitial fluid proteomics defines cell type-specific immune regulation in atopic dermatitis
10.1016/j.jaci.2020.03.041 · 2020 · External reference
IL-4 impairs wound healing potential in the skin by repressing fibronectin expression
10.1016/j.jaci.2016.07.012 · 2017 · External reference
Atopic Dermatitis Is an IL-13-Dominant Disease with Greater Molecular Heterogeneity Compared to Psoriasis
10.1016/j.jid.2018.12.018 · 2019 · External reference
An Integrated Model of Atopic Dermatitis Biomarkers Highlights the Systemic Nature of the Disease
10.1016/j.jid.2016.09.037 · 2017 · External reference
Emerging functions of amphiregulin in orchestrating immunity, inflammation, and tissue repair
10.1016/j.immuni.2015.01.020 · 2015 · External reference
