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References from The interplay of the lncRNA AFAP1-AS1 and the soluble immune checkpoint proteins sPD-1/sPD-L1 in breast cancer: tumor–immune cross-talk. Local targets link to admitted publications; unresolved targets remain external evidence.
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lncRNA AFAP1-AS1 promotes triple negative breast cancer cell proliferation and invasion via targeting miR-145 to regulate MTH1 expression
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LncRNA AFAP1-AS1 promotes the progression of colorectal cancer through miR-195-5p and WISP1
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Expression analysis of AFAP1-AS1 and AFAP1 in breast cancer
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Up-regulated lncRNA AFAP1-AS1 indicates a poor prognosis and promotes carcinogenesis of breast cancer
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Long noncoding RNA AFAP1-AS1 promotes tumor progression and invasion by regulating the miR-2110/Sp1 axis in triple-negative breast cancer
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LncRNA AFAP1-AS1 knockdown represses cell proliferation, migration, and induced apoptosis in breast cancer by downregulating SEPT2 via sponging miR-497-5p
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SP1-induced upregulation of LncRNA AFAP1-AS1 promotes tumor progression in triple-negative breast cancer by regulating mTOR pathway
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FAP positive cancer-associated fibroblasts promote tumor progression and radioresistance in esophageal squamous cell carcinoma by transferring exosomal lncRNA AFAP1-AS1
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Long non-coding RNA AFAP1-AS1 accelerates lung cancer cells migration and invasion by interacting with SNIP1 to upregulate c-Myc
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