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References from The mitochondrial deacetylase SIRT3 is nonessential for cyclophilin D-dependent procoagulant platelet formation in mice. Local targets link to admitted publications; unresolved targets remain external evidence.
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Mitochondrial hyperacetylation in the failing hearts of obese patients mediated partly by a reduction in SIRT3: the involvement of the mitochondrial permeability transition pore
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Indirubin and NAD(+) prevent mitochondrial ischaemia/reperfusion damage in fatty livers
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