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References from Single‐Cell <scp>RNA</scp> Sequencing‐Based Identification of Novel Podocyte Injury‐Associated Genes From Isolated Glomeruli of Diabetic <scp>db/db</scp> Mice. Local targets link to admitted publications; unresolved targets remain external evidence.
Podocyte Loss and Progressive Glomerular Injury in Type II Diabetes
10.1172/jci119163 · 1997 · External reference
Progression of Nephropathy in Type 2 Diabetic Patients
10.1111/j.1523-1755.2004.00925.x · 2004 · External reference
Podocyte Number in Normotensive Type 1 Diabetic Patients With Albuminuria
10.2337/diabetes.51.10.3083 · 2002 · External reference
Biochemistry and Molecular Cell Biology of Diabetic Complications
10.1038/414813a · 2001 · External reference
The Pathobiology of Diabetic Complications: A Unifying Mechanism
10.2337/diabetes.54.6.1615 · 2005 · External reference
Single‐Cell RNA Profiling of Glomerular Cells Shows Dynamic Changes in Experimental Diabetic Kidney Disease
10.1681/asn.2018090896 · 2019 · External reference
Single‐Cell Transcriptomics Reveals a Mechanosensitive Injury Signaling Pathway in Early Diabetic Nephropathy
10.1186/s13073-022-01145-4 · 2023 · External reference
Single‐Cell Transcriptomic Profiles in the Pathophysiology Within the Microenvironment of Early Diabetic Kidney Disease
10.1038/s41419-023-05947-1 · 2023 · External reference
Integrative ATAC‐Seq and RNA‐Seq Analysis Associated With Diabetic Nephropathy and Identification of Novel Targets for Treatment by Dapagliflozin
10.1002/cbf.3943 · 2024 · External reference
Unified Mouse and Human Kidney Single‐Cell Expression Atlas Reveal Commonalities and Differences in Disease States
10.1681/asn.0000000000000217 · 2023 · External reference
Single‐Cell Transcriptome Profiling of the Kidney Glomerulus Identifies Key Cell Types and Reactions to Injury
10.1681/asn.2020020220 · 2020 · External reference
ARP3 Controls the Podocyte Architecture at the Kidney Filtration Barrier
10.1016/j.devcel.2018.11.011 · 2018 · External reference
A Conditionally Immortalized Human Podocyte Cell Line Demonstrating Nephrin and Podocin Expression
10.1681/asn.v133630 · 2002 · External reference
Fatty Acids Are Novel Nutrient Factors to Regulate mTORC1 Lysosomal Localization and Apoptosis in Podocytes
10.1016/j.bbadis.2014.04.001 · 2014 · External reference
Impaired Podocyte Autophagy Exacerbates Proteinuria in Diabetic Nephropathy
10.2337/db15-0473 · 2016 · External reference
Neutralization of TGF‐Beta by Anti‐TGF‐Beta Antibody Attenuates Kidney Hypertrophy and the Enhanced Extracellular Matrix Gene Expression in STZ‐Induced Diabetic Mice
10.2337/diab.45.4.522 · 1996 · External reference
Glomerular‐Specific Alterations of VEGF‐A Expression Lead to Distinct Congenital and Acquired Renal Diseases
10.1172/jci17423 · 2003 · External reference
Operational Redundancy in Axon Guidance Through the Multifunctional Receptor Robo3 and Its Ligand NELL2
10.1126/science.aad2615 · 2015 · External reference
NELL2‐Mediated Lumicrine Signaling Through OVCH2 Is Required for Male Fertility
10.1126/science.aay5134 · 2020 · External reference
DACH1 Protects Podocytes From Experimental Diabetic Injury and Modulates PTIP‐H3K4Me3 Activity
10.1172/jci141279 · 2021 · External reference
NELL1 Membranous Nephropathy: Clinical Associations Provide Mechanistic Clues
10.3389/fneph.2024.1323432 · 2024 · External reference
The OPCML Tumor Suppressor Functions as a Cell Surface Repressor‐Adaptor, Negatively Regulating Receptor Tyrosine Kinases in Epithelial Ovarian Cancer
10.1158/2159-8290.cd-11-0256 · 2012 · External reference
OPCML at 11q25 Is Epigenetically Inactivated and Has Tumor‐Suppressor Function in Epithelial Ovarian Cancer
10.1038/ng1183 · 2003 · External reference
Plasma Adrenomedullin Levels in Patients With Non‐Insulin Dependent Diabetes Mellitus: Close Relationships With Diabetic Complications
10.1507/endocrj.45.241 · 1998 · External reference
Relationship Between Plasma Adrenomedullin Levels and Metabolic Control, Risk Factors, and Diabetic Microangiopathy in Patients With Type 2 Diabetes
10.2337/diacare.23.6.864a · 2000 · External reference
Dhrs3 Protein Attenuates Retinoic Acid Signaling and Is Required for Early Embryonic Patterning
10.1074/jbc.m113.514984 · 2013 · External reference
The Retinaldehyde Reductase DHRS3 Is Essential for Preventing the Formation of Excess Retinoic Acid During Embryonic Development
10.1096/fj.13-227967 · 2013 · External reference
p53‐Inducible DHRS3 Is an Endoplasmic Reticulum Protein Associated With Lipid Droplet Accumulation
10.1074/jbc.m111.254227 · 2011 · External reference
The Lipid Droplet Protein DHRS3 Is a Regulator of Melanoma Cell State
10.1111/pcmr.13208 · 2025 · External reference
ATRA Induces Podocyte Differentiation and Alters Nephrin and Podocin Expression In Vitro and In Vivo
10.1111/j.1523-1755.2005.00387.x · 2005 · External reference
Retinoic Acid Improves Nephrotoxic Serum‐Induced Glomerulonephritis Through Activation of Podocyte Retinoic Acid Receptor α
10.1016/j.kint.2017.04.026 · 2017 · External reference
Integrative ATAC‐Seq and RNA‐Seq Analysis Associated With Diabetic Nephropathy and Identification of Novel Targets for Treatment by Dapagliflozin
10.1002/cbf.3943 · ExternalCitation · doi-reference
Fatty Acids Are Novel Nutrient Factors to Regulate mTORC1 Lysosomal Localization and Apoptosis in Podocytes
10.1016/j.bbadis.2014.04.001 · ExternalCitation · doi-reference
ARP3 Controls the Podocyte Architecture at the Kidney Filtration Barrier
10.1016/j.devcel.2018.11.011 · ExternalCitation · doi-reference
Retinoic Acid Improves Nephrotoxic Serum‐Induced Glomerulonephritis Through Activation of Podocyte Retinoic Acid Receptor α
10.1016/j.kint.2017.04.026 · ExternalCitation · doi-reference
Biochemistry and Molecular Cell Biology of Diabetic Complications
10.1038/414813a · ExternalCitation · doi-reference
OPCML at 11q25 Is Epigenetically Inactivated and Has Tumor‐Suppressor Function in Epithelial Ovarian Cancer
10.1038/ng1183 · ExternalCitation · doi-reference
Single‐Cell Transcriptomic Profiles in the Pathophysiology Within the Microenvironment of Early Diabetic Kidney Disease
10.1038/s41419-023-05947-1 · ExternalCitation · doi-reference
p53‐Inducible DHRS3 Is an Endoplasmic Reticulum Protein Associated With Lipid Droplet Accumulation
10.1074/jbc.m111.254227 · ExternalCitation · doi-reference
Dhrs3 Protein Attenuates Retinoic Acid Signaling and Is Required for Early Embryonic Patterning
10.1074/jbc.m113.514984 · ExternalCitation · doi-reference
The Retinaldehyde Reductase DHRS3 Is Essential for Preventing the Formation of Excess Retinoic Acid During Embryonic Development
10.1096/fj.13-227967 · ExternalCitation · doi-reference
Progression of Nephropathy in Type 2 Diabetic Patients
10.1111/j.1523-1755.2004.00925.x · ExternalCitation · doi-reference
ATRA Induces Podocyte Differentiation and Alters Nephrin and Podocin Expression In Vitro and In Vivo
10.1111/j.1523-1755.2005.00387.x · ExternalCitation · doi-reference
The Lipid Droplet Protein DHRS3 Is a Regulator of Melanoma Cell State
10.1111/pcmr.13208 · ExternalCitation · doi-reference
Operational Redundancy in Axon Guidance Through the Multifunctional Receptor Robo3 and Its Ligand NELL2
10.1126/science.aad2615 · ExternalCitation · doi-reference
NELL2‐Mediated Lumicrine Signaling Through OVCH2 Is Required for Male Fertility
10.1126/science.aay5134 · ExternalCitation · doi-reference
The OPCML Tumor Suppressor Functions as a Cell Surface Repressor‐Adaptor, Negatively Regulating Receptor Tyrosine Kinases in Epithelial Ovarian Cancer
10.1158/2159-8290.cd-11-0256 · ExternalCitation · doi-reference
Podocyte Loss and Progressive Glomerular Injury in Type II Diabetes
10.1172/jci119163 · ExternalCitation · doi-reference
DACH1 Protects Podocytes From Experimental Diabetic Injury and Modulates PTIP‐H3K4Me3 Activity
10.1172/jci141279 · ExternalCitation · doi-reference
Glomerular‐Specific Alterations of VEGF‐A Expression Lead to Distinct Congenital and Acquired Renal Diseases
10.1172/jci17423 · ExternalCitation · doi-reference
Single‐Cell Transcriptomics Reveals a Mechanosensitive Injury Signaling Pathway in Early Diabetic Nephropathy
10.1186/s13073-022-01145-4 · ExternalCitation · doi-reference
Plasma Adrenomedullin Levels in Patients With Non‐Insulin Dependent Diabetes Mellitus: Close Relationships With Diabetic Complications
10.1507/endocrj.45.241 · ExternalCitation · doi-reference
Unified Mouse and Human Kidney Single‐Cell Expression Atlas Reveal Commonalities and Differences in Disease States
10.1681/asn.0000000000000217 · ExternalCitation · doi-reference
Single‐Cell RNA Profiling of Glomerular Cells Shows Dynamic Changes in Experimental Diabetic Kidney Disease
10.1681/asn.2018090896 · ExternalCitation · doi-reference
Single‐Cell Transcriptome Profiling of the Kidney Glomerulus Identifies Key Cell Types and Reactions to Injury
10.1681/asn.2020020220 · ExternalCitation · doi-reference
A Conditionally Immortalized Human Podocyte Cell Line Demonstrating Nephrin and Podocin Expression
10.1681/asn.v133630 · ExternalCitation · doi-reference
Impaired Podocyte Autophagy Exacerbates Proteinuria in Diabetic Nephropathy
10.2337/db15-0473 · ExternalCitation · doi-reference
Neutralization of TGF‐Beta by Anti‐TGF‐Beta Antibody Attenuates Kidney Hypertrophy and the Enhanced Extracellular Matrix Gene Expression in STZ‐Induced Diabetic Mice
10.2337/diab.45.4.522 · ExternalCitation · doi-reference
Podocyte Number in Normotensive Type 1 Diabetic Patients With Albuminuria
10.2337/diabetes.51.10.3083 · ExternalCitation · doi-reference
The Pathobiology of Diabetic Complications: A Unifying Mechanism
10.2337/diabetes.54.6.1615 · ExternalCitation · doi-reference
Relationship Between Plasma Adrenomedullin Levels and Metabolic Control, Risk Factors, and Diabetic Microangiopathy in Patients With Type 2 Diabetes
10.2337/diacare.23.6.864a · ExternalCitation · doi-reference
NELL1 Membranous Nephropathy: Clinical Associations Provide Mechanistic Clues
10.3389/fneph.2024.1323432 · ExternalCitation · doi-reference
