References

A comprehensive review of the genetics of juvenile idiopathic arthritis.

10.1186/1546-0096-6-11 · 2008 · External reference

Disease course and outcome of juvenile rheumatoid arthritis in a multicenter cohort.

2002 · External reference

Juvenile idiopathic arthritis.

10.1542/pir.2022-005623 · 2023 · External reference

The genetics of juvenile idiopathic arthritis: what is new in 2010?

10.1007/s11926-010-0087-0 · 2010 · External reference

An overview of genetics of paediatric rheumatic diseases.

10.1016/j.berh.2009.08.001 · 2009 · External reference

Subclinical cardiovascular risk signs in adults with juvenile idiopathic arthritis in sustained remission.

10.1186/s12969-020-00448-3 · 2020 · External reference

Tumor necrosis factor-α -308 A/G gene polymorphism in children with juvenile idiopathic arthritis: relation to disease activity, damage, and functional status.

10.1007/s10067-017-3719-1 · 2017 · External reference

Influence of interleukin-4 gene polymorphisms and interleukin-4 serum level on susceptibility and severity of rheumatoid arthritis in Egyptian population.

10.1016/j.cyto.2013.01.001 · 2013 · External reference

The regulation of immune tolerance by FOXP3.

10.1038/nri.2017.75 · 2017 · External reference

Cytokine storm-definition, causes, and implications.

10.3390/ijms231911740 · 2022 · External reference

Cytokines: from clinical significance to quantification.

10.1002/advs.202004433 · 2021 · External reference

Mendelian randomization.

10.1007/978-1-4939-7274-6_29 · 2017 · External reference

Mendelian randomization analysis with multiple genetic variants using summarized data.

10.1002/gepi.21758 · 2013 · External reference

Using multiple genetic variants as instrumental variables for modifiable risk factors.

10.1177/0962280210394459 · 2012 · External reference

Using published data in Mendelian randomization: a blueprint for efficient identification of causal risk factors.

10.1007/s10654-015-0011-z · 2015 · External reference

Mendelian randomization: genetic anchors for causal inference in epidemiological studies.

10.1093/hmg/ddu328 · 2014 · External reference

The MR-Base platform supports systematic causal inference across the human phenome.

10.7554/elife.34408 · 2018 · External reference

Avoiding bias from weak instruments in Mendelian randomization studies.

10.1093/ije/dyr036 · 2011 · External reference

Guidelines for performing Mendelian randomization investigations: update for summer 2023.

10.12688/wellcomeopenres.15555.1 · 2019 · External reference

A comparison of robust Mendelian randomization methods using summary data.

10.1002/gepi.22295 · 2020 · External reference

Mendelian randomization with invalid instruments: effect estimation and bias detection through Egger regression.

10.1093/ije/dyv080 · 2015 · External reference

Consistent estimation in Mendelian randomization with some invalid instruments using a weighted median estimator.

10.1002/gepi.21965 · 2016 · External reference

Detecting pleiotropy in Mendelian randomisation studies with summary data and a continuous outcome.

10.1002/sim.6522 · 2015 · External reference

Sensitivity analyses for robust causal inference from Mendelian randomization analyses with multiple genetic variants.

10.1097/ede.0000000000000559 · 2017 · External reference

Orienting the causal relationship between imprecisely measured traits using GWAS summary data.

10.1371/journal.pgen.1007081 · 2017 · External reference

CCL19 and CCL21 induce a potent proinflammatory differentiation program in licensed dendritic cells.

10.1016/j.immuni.2005.02.010 · 2005 · External reference

CCL19 and CCR7 expression, signaling pathways, and adjuvant functions in viral infection and prevention.

10.3389/fcell.2019.00212 · 2019 · External reference

Real-life 10-year retention rate of first-line anti-TNF drugs for inflammatory arthritides in adult- and juvenile-onset populations: similarities and differences.

10.1007/s10067-017-3712-8 · 2017 · External reference

The CC chemokine CK beta-11/MIP-3 beta/ELC/Exodus 3 mediates tumor rejection of murine breast cancer cells through NK cells.

10.4049/jimmunol.164.8.4025 · 2000 · External reference

Body mass index, physical activity, and serum markers of inflammation, immunity, and insulin resistance.

10.1158/1055-9965.epi-14-0699-t · 2014 · External reference

FGF19 as a postprandial, insulin-independent activator of hepatic protein and glycogen synthesis.

10.1126/science.1198363 · 2011 · External reference

Mechanism of tissue-specific farnesoid X receptor in suppressing the expression of genes in bile-acid synthesis in mice.

10.1002/hep.25740 · 2012 · External reference

A nontumorigenic variant of FGF19 treats cholestatic liver diseases.

10.1126/scitranslmed.3009098 · 2014 · External reference

Endocrine fibroblast growth factors 15/19 and 21: from feast to famine.

10.1101/gad.184788.111 · 2012 · External reference

Minireview: roles of fibroblast growth factors 19 and 21 in metabolic regulation and chronic diseases.

10.1210/me.2015-1155 · 2015 · External reference

Fibroblast growth factor 19 alleviates palmitic acid-induced mitochondrial dysfunction and oxidative stress via the AMPK/PGC-1α pathway in skeletal muscle.

10.1016/j.bbrc.2020.04.002 · 2020 · External reference

FGF19 action in the brain induces insulin-independent glucose lowering.

10.1172/jci70710 · 2013 · External reference

Monocyte chemotactic protein-3: possible involvement in apical periodontitis chemotaxis.

10.1111/j.1365-2591.2010.01764.x · 2010 · External reference

Reduced degradation of the chemokine MCP-3 by matrix metalloproteinase-2 exacerbates myocardial inflammation in experimental viral cardiomyopathy.

10.1161/circulationaha.111.035964 · 2011 · External reference

Critical roles for CCR2 and MCP-3 in monocyte mobilization from bone marrow and recruitment to inflammatory sites.

10.1172/jci29919 · 2007 · External reference