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References from Myocardial stretch‐induced ROS production is enhanced prior to the development of heart failure under TAC‐induced pressure overload. Local targets link to admitted publications; unresolved targets remain external evidence.
Activation of MMP‐2 as a key event in oxidative stress injury to the heart
2009 · External reference
Biophysical properties and functional consequences of reactive oxygen species (ROS)‐induced ROS release in intact myocardium
10.1113/jphysiol.2011.214239 · 2011 · External reference
Role of purinergic activation and TRPC3 channels in the Frank‐Starling mechanism
10.1016/j.jphyss.2025.100052 · 2026 · External reference
The role of oxidative stress in cardiac disease: From physiological response to injury factor
2020 · External reference
Activation of the cardiac ryanodine receptor by sulfhydryl oxidation is modified by Mg2+ and ATP
10.1007/s002329900365 · 1998 · External reference
Cross‐talk between NADPH oxidase and mitochondria: Role in ROS signaling and angiogenesis
10.3390/cells9081849 · 2020 · External reference
Involvement of the nicotinamide adenosine dinucleotide phosphate oxidase isoform Nox2 in cardiac contractile dysfunction occurring in response to pressure overload
10.1016/j.jacc.2005.09.051 · 2006 · External reference
Novel aspects of ROS signalling in heart failure
10.1007/s00395-013-0359-8 · 2013 · External reference
Mechano‐sensitivity of mitochondrial function in mouse cardiac myocytes
10.1016/j.pbiomolbio.2017.05.015 · 2017 · External reference
Axial stretch enhances sarcoplasmic reticulum Ca2+ leak and cellular Ca2+ reuptake in guinea pig ventricular myocytes: Experiments and models
10.1016/j.pbiomolbio.2008.02.012 · 2008 · External reference
Activation of intracellular matrix metalloproteinase‐2 by reactive oxygen–nitrogen species: Consequences and therapeutic strategies in the heart
10.1016/j.abb.2013.09.019 · 2013 · External reference
Stretch‐induced reactive oxygen species contribute to the Frank–Starling mechanism
10.1113/jp284283 · 2024 · External reference
Lack of tissue inhibitor of metalloproteinases 2 leads to exacerbated left ventricular dysfunction and adverse extracellular matrix remodeling in response to biomechanical stress
10.1161/circulationaha.111.030338 · 2011 · External reference
Matrix metalloproteinase‐2 and myocardial oxidative stress injury: Beyond the matrix
10.1093/cvr/cvp268 · 2010 · External reference
Gender difference in oxidative stress: A new look at the mechanisms for cardiovascular diseases
10.1111/jcmm.13038 · 2017 · External reference
Reactive oxygen species detection with fluorescent probes: Limitations and recommendations beyond DCFH‐DA
10.1021/acs.jmedchem.5c03494 · 2026 · External reference
Mitochondrial creatine kinase attenuates pathologic remodeling in heart failure
2022 · External reference
ROS‐induced ROS release orchestrated by Nox4, Nox2, and mitochondria in VEGF signaling and angiogenesis
10.1152/ajpcell.00346.2016 · 2017 · External reference
TRPC3 positively regulates reactive oxygen species driving maladaptive cardiac remodeling
10.1038/srep37001 · 2016 · External reference
Reactive oxygen species and excitation‐contraction coupling in the context of cardiac pathology
10.1016/j.yjmcc.2014.03.001 · 2014 · External reference
Interplay of matrix metalloproteinases, tissue inhibitors of metalloproteinases and their regulators in cardiac matrix remodeling
10.1016/s0008-6363(00)00003-1 · 2000 · External reference
Stachydrine hydrochloride regulates the NOX2‐ROS‐signaling axis in pressure‐overload‐induced heart failure
10.3390/ijms241814369 · 2023 · External reference
Sex hormone regulation of proteins modulating mitochondrial metabolism, dynamics and inter‐organellar cross talk in cardiovascular disease
10.3389/fcell.2020.610516 · 2021 · External reference
Sulfhydryl oxidation modifies the calcium dependence of ryanodine‐ sensitive calcium channels of excitable cells
10.1016/s0006-3495(98)77840-3 · 1998 · External reference
Short overview of ROS as cell function regulators and their implications in therapy concepts
10.3390/cells8080793 · 2019 · External reference
Tissue inhibitor of metalloproteinases (TIMPs) in heart failure
10.1007/s10741-011-9266-y · 2012 · External reference
P2Y6 receptor‐Galpha12/13 signalling in cardiomyocytes triggers pressure overload‐induced cardiac fibrosis
10.1038/emboj.2008.237 · 2008 · External reference
TRPC3‐GEF‐H1 axis mediates pressure overload‐induced cardiac fibrosis
10.1038/srep39383 · 2016 · External reference
Loss of p47phox subunit enhances susceptibility to biomechanical stress and heart failure because of dysregulation of cortactin and actin filaments
10.1161/circresaha.111.300299 · 2013 · External reference
X‐ROS signaling in the heart and skeletal muscle: Stretch‐dependent local ROS regulates [Ca2+]i
10.1016/j.yjmcc.2012.11.011 · 2013 · External reference
X‐ROS signalling is enhanced and graded by cyclic cardiomyocyte stretch
10.1093/cvr/cvt066 · 2013 · External reference
X‐ROS signaling: Rapid mechano‐chemo transduction in heart
10.1126/science.1202768 · 2011 · External reference
Distinct phenotypes induced by three degrees of transverse aortic constriction in mice
10.1038/s41598-019-42209-7 · 2019 · External reference
Dipeptidyl‐peptidase IV inhibition improves pathophysiology of heart failure and increases survival rate in pressure‐overloaded mice
10.1152/ajpheart.00454.2012 · 2013 · External reference
Imaging ROS signaling in cells and animals
10.1007/s00109-013-1067-4 · 2013 · External reference
Activation of the cardiac calcium release channel (ryanodine receptor) by poly‐S‐nitrosylation
10.1126/science.279.5348.234 · 1998 · External reference
TRPC3 participates in angiotensin II type 1 receptor‐dependent stress‐induced slow increase in intracellular Ca2+ concentration in mouse cardiomyocytes
10.1007/s12576-016-0519-3 · 2018 · External reference
