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References from Low-dose mixtures of dietary nutrients ameliorate behavioral deficits in multiple mouse models of autism. Local targets link to admitted publications; unresolved targets remain external evidence.
The diagnosis of autism: from kanner to DSM-III to DSM-5 and beyond
10.1007/s10803-021-04904-1 · 2021 · External reference
Recent advances in autism research as reflected in DSM-5 criteria for autism spectrum disorder
10.1146/annurev-clinpsy-032814-112745 · 2015 · External reference
Neuropsychopathology of autism spectrum disorder: complex interplay of genetic, epigenetic, and environmental factors
10.1007/978-3-030-30402-7_4 · 2020 · External reference
Autism risk factors: genes, environment, and gene-environment interactions
10.31887/dcns.2012.14.3/pchaste · 2012 · External reference
Synaptic, transcriptional and chromatin genes disrupted in autism
10.1038/nature13772 · 2014 · External reference
Autism spectrum disorders: developmental disconnection syndromes
10.1016/j.conb.2007.01.009 · 2007 · External reference
Signaling in autism: relevance to nutrients and sex
10.1016/j.conb.2024.102962 · 2025 · External reference
Nutritional interventions for autism spectrum disorder
10.1093/nutrit/nuz092 · 2020 · External reference
Autism-related dietary preferences mediate autism-gut microbiome associations
2021 · External reference
Exercise and hippocampal memory systems
10.1016/j.tics.2019.01.006 · 2019 · External reference
BCKDK deficiency: a treatable neurodevelopmental disease amenable to newborn screening
10.1093/brain/awad010 · 2023 · External reference
Zinc deficiency dysregulates the synaptic ProSAP/Shank scaffold and might contribute to autism spectrum disorders
10.1093/brain/awt303 · 2014 · External reference
Phase separation and zinc-induced transition modulate synaptic distribution and association of autism-linked CTTNBP2 and SHANK3
10.1038/s41467-022-30353-0 · 2022 · External reference
CTTNBP2 controls synaptic expression of zinc-related autism-associated proteins and regulates synapse formation and autism-like behaviors
10.1016/j.celrep.2020.107700 · 2020 · External reference
VCP and ATL1 regulate endoplasmic reticulum and protein synthesis for dendritic spine formation
10.1038/ncomms11020 · 2016 · External reference
Vcp overexpression and leucine supplementation increase protein synthesis and improve fear memory and social interaction of Nf1 mutant mice
10.1016/j.celrep.2020.107835 · 2020 · External reference
Social behaviors and contextual memory of Vcp mutant mice are sensitive to nutrition and can be ameliorated by amino acid supplementation
10.1016/j.isci.2020.101949 · 2020 · External reference
Sex bias in social deficits, neural circuits and nutrient demand in Cttnbp2 autism models
10.1093/brain/awac429 · 2023 · External reference
Trans-synaptic zinc mobilization improves social interaction in two mouse models of autism through NMDAR activation
10.1038/ncomms8168 · 2015 · External reference
Zinc signal in brain diseases
10.3390/ijms18122506 · 2017 · External reference
An architectural framework that may lie at the core of the postsynaptic density
10.1126/science.1118995 · 2006 · External reference
Sex-biased zinc responses modulate ribosomal protein expression, protein synthesis and social defects in Cttnbp2 mutant mice
10.1016/j.nbd.2025.106932 · 2025 · External reference
Influence of maternal zinc supplementation on the development of autism-associated behavioural and synaptic deficits in offspring Shank3-knockout mice
10.1186/s13041-020-00650-0 · 2020 · External reference
In vitro zinc supplementation alters synaptic deficits caused by autism spectrum disorder-associated Shank2 point mutations in hippocampal neurons
10.1186/s13041-021-00809-3 · 2021 · External reference
Dietary zinc supplementation prevents autism related behaviors and striatal synaptic dysfunction in Shank3 exon 13-16 mutant mice
10.3389/fncel.2018.00374 · 2018 · External reference
Autism-linked mutations of CTTNBP2 reduce social interaction and impair dendritic spine formation via diverse mechanisms
10.1186/s40478-020-01053-x · 2020 · External reference
Dietary zinc supplementation rescues fear-based learning and synaptic function in the Tbr1+/- mouse model of autism spectrum disorders
10.1186/s13229-022-00494-6 · 2022 · External reference
D-serine released by astrocytes in brainstem regulates breathing response to CO2 levels
10.1038/s41467-017-00960-3 · 2017 · External reference
D-Serine as the gatekeeper of NMDA receptor activity: implications for the pharmacologic management of anxiety disorders
10.1038/s41398-020-00870-x · 2020 · External reference
D-serine is an endogenous ligand for the glycine site of the N-methyl-D-aspartate receptor
10.1073/pnas.97.9.4926 · 2000 · External reference
L-Serine dietary supplementation is associated with clinical improvement of loss-of-function GRIN2B-related pediatric encephalopathy
10.1126/scisignal.aaw0936 · 2019 · External reference
Mutations in BCKD-kinase lead to a potentially treatable form of autism with epilepsy
10.1126/science.1224631 · 2012 · External reference
Impaired amino acid transport at the blood brain barrier is a cause of autism spectrum disorder
10.1016/j.cell.2016.11.013 · 2016 · External reference
Sestrin2 is a leucine sensor for the mTORC1 pathway
10.1126/science.aab2674 · 2016 · External reference
SAR1B senses leucine levels to regulate mTORC1 signalling
10.1038/s41586-021-03768-w · 2021 · External reference
The molecular basis of nutrient sensing and signalling by mTORC1 in metabolism regulation and disease
10.1038/s41580-023-00641-8 · 2023 · External reference
The central role of mTORC1 in amino acid sensing
10.1158/0008-5472.can-21-4403 · 2022 · External reference
Valosin-containing protein and neurofibromin interact to regulate dendritic spine density
10.1172/jci45677 · 2011 · External reference
Tbr1 haploinsufficiency impairs amygdalar axonal projections and results in cognitive abnormality
10.1038/nn.3626 · 2014 · External reference
T-Brain-1—a potential master regulator in autism spectrum disorders
10.1002/aur.1456 · 2015 · External reference
Interhemispheric connectivity potentiates the basolateral amygdalae and regulates social interaction and memory
2019 · External reference
Haploinsufficiency of autism causative gene Tbr1 impairs olfactory discrimination and neuronal activation of the olfactory system in mice
10.1186/s13229-019-0257-5 · 2019 · External reference
Deep brain stimulation of the Tbr1-deficient mouse model of autism spectrum disorder at the basolateral amygdala alters amygdalar connectivity, whole-brain synchronization, and social behaviors
2024 · External reference
Cortactin-binding protein 2 modulates the mobility of cortactin and regulates dendritic spine formation and maintenance
10.1523/jneurosci.4405-11.2012 · 2012 · External reference
Cortactin-binding protein 2 increases microtubule stability and regulates dendritic arborization
2014 · External reference
PyWGCNA: a Python package for weighted gene co-expression network analysis
10.1093/bioinformatics/btad415 · 2023 · External reference
State-dependent encoding of exploratory behaviour in the amygdala
10.1038/s41586-021-03301-z · 2021 · External reference
Distinct and dynamic ON and OFF neural ensembles in the prefrontal cortex code social exploration
10.1016/j.neuron.2018.08.043 · 2018 · External reference
Infant visual brain development and inherited genetic liability in autism
10.1176/appi.ajp.21101002 · 2022 · External reference
Correlated neural activity and encoding of behavior across brains of socially interacting animals
10.1016/j.cell.2019.05.022 · 2019 · External reference
De novo TBR1 variants cause a neurocognitive phenotype with ID and autistic traits: report of 25 new individuals and review of the literature
10.1038/s41431-020-0571-6 · 2020 · External reference
The evolutionarily conserved function of TBR1 in controlling the size of anterior commissure in human and mouse brains
10.1038/s41431-020-0621-0 · 2020 · External reference
Neonatal Tbr1 dosage controls cortical layer 6 connectivity
10.1016/j.neuron.2018.09.027 · 2018 · External reference
Absorption mechanisms of iron, copper, and zinc: an overview
10.3177/jnsv.64.1 · 2018 · External reference
The families of zinc (SLC30 and SLC39) and copper (SLC31) transporters
10.1016/b978-0-12-800223-0.00009-8 · 2014 · External reference
Branched-chain amino acids and brain function
10.1093/jn/135.6.1539s · 2005 · External reference
Side effects of amino acid supplements
10.33549/physiolres.934790 · 2022 · External reference
Transcriptional modification by a CASK-interacting nucleosome assembly protein
10.1016/s0896-6273(04)00139-4 · 2004 · External reference
Identification of Tbr-1/CASK complex target genes in neurons
10.1111/j.1471-4159.2004.02845.x · 2004 · External reference
The ProteomeXchange consortium at 10 years: 2023 update
10.1093/nar/gkac1040 · 2023 · External reference
The PRIDE database at 20 years: 2025 update
10.1093/nar/gkae1011 · 2025 · External reference
WGCNA: an R package for weighted correlation network analysis
10.1186/1471-2105-9-559 · 2008 · External reference
CASK associates with glutamate receptor interacting protein and signaling molecules
10.1016/j.bbrc.2006.10.113 · 2006 · External reference
Differential K+ channel clustering activity of PSD-95 and SAP97, two related membrane-associated putative guanylate kinases
10.1016/0028-3908(96)00093-7 · 1996 · External reference
Targeted deletion of CASK-interacting nucleosome assembly protein causes higher locomotor and exploratory activities
10.1159/000327819 · 2011 · External reference
Sarm1, a neuronal inflammatory regulator, controls social interaction, associative memory and cognitive flexibility in mice
10.1016/j.bbi.2013.12.002 · 2014 · External reference
CaImAn an open source tool for scalable calcium imaging data analysis
10.7554/elife.38173 · 2019 · External reference
High-performance calcium sensors for imaging activity in neuronal populations and microcompartments
10.1038/s41592-019-0435-6 · 2019 · External reference
