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References from Novel insights into RAGE signaling pathways during the progression of amyotrophic lateral sclerosis in RAGE-deficient SOD1 G93A mice. Local targets link to admitted publications; unresolved targets remain external evidence.
Role of RAGE in the Pathogenesis of Neurological Disorders
10.1007/s12264-022-00878-x · 2022 · External reference
RAGE axis in neuroinflammation, neurodegeneration and its emerging role in the pathogenesis of amyotrophic lateral sclerosis
10.1016/j.neubiorev.2015.12.006 · 2016 · External reference
Receptor for Advanced Glycation End Products and its Inflammatory Ligands are Upregulated in Amyotrophic Lateral Sclerosis
10.3389/fncel.2015.00485 · 2015 · External reference
Soluble RAGE Treatment Delays Progression of Amyotrophic Lateral Sclerosis in SOD1
2016 · External reference
Risk Factors and Emerging Therapies in Amyotrophic Lateral Sclerosis
10.3390/ijms20112616 · 2019 · External reference
The Involvement of RAGE and Its Ligands during Progression of ALS in SOD1 G93A Transgenic Mice
10.3390/ijms23042184 · 2022 · External reference
Absence of Receptor for Advanced Glycation End Product (RAGE) Reduces Inflammation and Extends Survival in the hSOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis
10.1007/s12035-020-02019-9 · 2020 · External reference
The Receptor for Advanced Glycation Endproducts (RAGE) and Mediation of Inflammatory Neurodegeneration
10.4172/2161-0460.1000421 · 2018 · External reference
Microglia RAGE exacerbates the progression of neurodegeneration within the SOD1G93A murine model of amyotrophic lateral sclerosis in a sex-dependent manner.
10.1186/s12974-021-02191-2 · 2021 · External reference
Novel insights into the nervous system affected by prolonged hyperglycemia
10.1007/s00109-023-02347-y · 2023 · External reference
3-Nitropropionic acid modifies neurotrophin mRNA expression in the mouse striatum: 18S-rRNA is a reliable control gene for studies of the striatum
10.1007/s12264-012-1259-x · 2012 · External reference
Bisphenol A-Induced changes in the enteric nervous system of the porcine duodenum
10.1016/j.neuro.2018.03.008 · 2018 · External reference
Comparing two small samples with an unstable, treatment-independent baseline
10.1016/j.jneumeth.2009.01.017 · 2009 · External reference
Dysregulation of Rac or Rho elicits death of motor neurons and activation of these GTPases is altered in the G93A mutant hSOD1 mouse model of amyotrophic lateral sclerosis
10.1016/j.nbd.2020.104743 · 2020 · External reference
Widespread immunoreactivity for neuronal nuclei in cultured human and rodent astrocytes
10.1111/j.1471-4159.2007.05043.x · 2008 · External reference
Peripheral Neuropathy Presents Similar Symptoms and Pathological Changes in Both High-Fat Diet and Pharmacologically Induced Pre- and Diabetic Mouse Models
2021 · External reference
Neuronal-glial communication perturbations in murine SOD1G93A spinal cord
10.1038/s42003-022-03128-y · 2022 · External reference
Therapeutic blockade of HMGB1 reduces early motor deficits, but not survival in the SOD1G93A mouse model of amyotrophic lateral sclerosis
10.1186/s12974-019-1435-2 · 2019 · External reference
Absence of toll-like receptor 4 (TLR4) extends survival in the hSOD1 G93A mouse model of amyotrophic lateral sclerosis
10.1186/s12974-015-0310-z · 2015 · External reference
The Astrocytic S100B Protein with Its Receptor RAGE Is Aberrantly Expressed in SOD1G93A Models, and Its Inhibition Decreases the Expression of Proinflammatory Genes
10.1155/2017/1626204 · 2017 · External reference
Small-molecule antagonism of the interaction of the RAGE cytoplasmic domain with DIAPH1 reduces diabetic complications in mice
10.1126/scitranslmed.abf7084 · 2021 · External reference
Small Molecule Inhibition of Ligand-Stimulated RAGE-DIAPH1 Signal Transduction
10.1038/srep22450 · 2016 · External reference
Effects of RAGE inhibition on the progression of the disease in hSOD1G93A ALS mice
10.1002/prp2.636 · 2020 · External reference
Widespread immunoreactivity for neuronal nuclei in cultured human and rodent astrocytes
10.1111/j.1471-4159.2007.05043.x · 2008 · External reference
GFAP positivity in neurons following traumatic brain injuries
10.1007/s00414-021-02568-1 · 2021 · External reference
Novel serum autoantibodies against ß-actin (ACTB) in amyotrophic lateral sclerosis
10.1080/21678421.2021.1885448 · 2021 · External reference
DNA damage as a mechanism of neurodegeneration in ALS and a contributor to astrocyte toxicity
10.1007/s00018-021-03872-0 · 2021 · External reference
Induction of glial fibrillary acidic protein expression in astrocytes by nitric oxide
10.1523/jneurosci.5480-05.2006 · 2006 · External reference
CRISPR/Cas9 Technology as an Emerging Tool for Targeting Amyotrophic Lateral Sclerosis (ALS)
10.3390/ijms19030906 · 2018 · External reference
Glial cells in amyotrophic lateral sclerosis
10.1155/2011/718987 · 2011 · External reference
Astrocytes and Microglia as Non-cell Autonomous Players in the Pathogenesis of ALS
10.5607/en.2016.25.5.233 · 2016 · External reference
Effect of transgene copy number on survival in the G93A SOD1 transgenic mouse model of ALS
10.1016/j.molbrainres.2004.07.002 · 2004 · External reference
Microglia centered pathogenesis in ALS: insights in cell interconnectivity.
10.3389/fncel.2014.00117 · 2014 · External reference
Microglial physiology: unique stimuli, specialized responses
10.1146/annurev.immunol.021908.132528 · 2009 · External reference
Glial-restricted precursors as potential candidates for ALS cell-replacement therapy
10.55782/ane-2014-1989 · 2014 · External reference
Stem cells for ALS: An overview of possible therapeutic approaches
10.1016/j.ijdevneu.2017.01.003 · 2017 · External reference
The receptor for advanced glycation end products and its ligands’ expression in OVE26 diabetic sciatic nerve during the development of length-dependent neuropathy
10.1111/neup.12852 · 2023 · External reference
β-Actin specifically controls cell growth, migration, and the G-actin pool
10.1091/mbc.e11-06-0582 · 2011 · External reference
Differential roles of α-, β-, and γ-actin in axon growth and collateral branch formation in motoneurons
10.1083/jcb.201604117 · 2017 · External reference
Neuroprotection by upregulation of the major histocompatibility complex class I (MHC I) in SOD1G93A mice
10.3389/fncel.2023.1211486 · 2023 · External reference
Delayed onset, immunomodulation, and lifespan improvement of SOD1G93A mice after intravenous injection of human mesenchymal stem cells derived from adipose tissue
10.1016/j.brainresbull.2022.06.008 · 2022 · External reference
