Abstract
No public abstract is available from the verified source records.
Contact and support
Need help, have a question, or want to contact the ResearchHub team?
© 2026 ResearchHub. Built for responsible scholarly connection.
Privacy-aware community infrastructurePeiqi Huang, Hongting Pan, Congzheng Qi, Yongqi Huang, Jinhui Li
Abstract
Authors
Institutions
No ROR-resolved institution is linked to this work yet.
Provenance
crossref
Confidence 100%
unpaywall
Confidence 95%
datacite
Confidence 0%
No local reference links have been materialized yet.
No local citing links have been materialized yet.
Temporal and geographic variability of bisphenol levels in humans: A systematic review and meta-analysis of international biomonitoring data
10.1016/j.envres.2024.120341 · 2025
Perinatal bisphenol A exposure promotes hyperactivity, lean body composition, and hormonal responses across the murine life course
10.1096/fj.12-223545 · 2013
Corrigendum to “Modulation of ceramides through nutrition: A new target in obesity and insulin resistance (Narrative Review)” [Clin Nutr ESPEN 71 (2026) 102836]
10.1016/j.clnesp.2025.102890 · 2026
Transfer of bisphenol A across the human placenta
2010
Transmaternal bisphenol A exposure accelerates diabetes type 1 development in NOD mice
10.1093/toxsci/kft242 · 2014
Bisphenol A in human placental and fetal liver tissues collected from Greater Montreal area (Quebec) during 1998-2008
10.1016/j.chemosphere.2012.05.003 · 2012
Adipose Tissue Dysfunction Determines Lipotoxicity and Triggers the Metabolic Syndrome: Current Challenges and Clinical Perspectives
10.1007/978-3-031-63657-8_8 · 2024
Targeting a ceramide double bond improves insulin resistance and hepatic steatosis
10.1126/science.aav3722 · 2019
The role of cord blood lipidomics in the associations of prenatal exposure to bisphenol A and its analogs with infant growth: A birth cohort study
10.1016/j.ecoenv.2025.119171 · 2025
Association between bisphenol A exposure and cardiometabolic outcomes: A longitudinal approach
10.1016/j.jhazmat.2024.135000 · 2024
The Role of Endocrine Disruptors Bisphenols and Phthalates in Obesity: Current Evidence, Perspectives and Controversies
10.3390/ijms25010675 · 2024
Endocrine-disrupting chemicals: an Endocrine Society scientific statement
10.1210/er.2009-0002 · 2009
Biomonitoring of bisphenols A, F, S in human milk and probabilistic risk assessment for breastfed infants
10.1016/j.scitotenv.2019.03.024 · 2019
Re-evaluation of the risks to public health related to the presence of bisphenol A (BPA) in foodstuffs
2023
Plasma ceramides containing saturated fatty acids are associated with risk of type 2 diabetes
10.1016/j.jlr.2021.100119 · 2021
Effects of Bisphenol A on the Risk of Developing Obesity
10.3390/nu16213740 · 2024
Weight-of-evidence evaluation of reproductive and developmental effects of low doses of bisphenol A
10.1080/10408440802157839 · 2009
Ceramide Synthase 5 Is Essential to Maintain C16:0-Ceramide Pools and Contributes to the Development of Diet-induced Obesity
10.1074/jbc.m115.691212 · 2016
The association between prenatal exposure to bisphenol A and offspring obesity: A systematic review
10.1016/j.envpol.2024.123391 · 2024
Urinary bisphenol A concentration and the risk of central obesity in Chinese adults: A prospective study
10.1111/1753-0407.12531 · 2018
Ceramide Acyl Chain Length and Its Relevance to Intracellular Lipid Regulation
10.3390/ijms23179697 · 2022
Urinary bisphenol A is associated with insulin resistance and obesity in reproductive-aged women
10.1111/cen.13270 · 2017
Low dose effect of in utero exposure to bisphenol A and diethylstilbestrol on female mouse reproduction
10.1016/s0890-6238(02)00006-0 · 2002
The combined effect of early-life Bisphenol A (BPA) exposure and post-weaning high-fat diet on the intestinal health of mice
10.1016/j.fct.2026.116233 · 2026
Lipid profiling identifies modifiable signatures of cardiometabolic risk in children and adolescents with obesity
10.1038/s41591-024-03279-x · 2025
Sex-dependent effects of developmental exposure to bisphenol A and ethinyl estradiol on metabolic parameters and voluntary physical activity
10.1017/s2040174415001488 · 2015
Human exposure to bisphenol A
10.1016/j.tox.2006.06.009 · 2006
Adipose Tissue and Endocrine-Disrupting Chemicals: Does Sex Matter?
10.3390/ijerph17249403 · 2020
Metabolic impact of endogenously produced estrogens by adipose tissue in females and males across the lifespan
10.3389/fendo.2025.1682231 · 2025
Association between bisphenol A exposure and adiposity measures in children
10.1097/md.0000000000041065 · 2024
Endocrine-disrupting chemicals in human adipose tissue and associations between exposure and obesity
10.1016/j.jes.2024.09.008 · 2025
Extraction and Analysis of Pan-metabolome Polar Metabolites by Ultra Performance Liquid Chromatography-Tandem Mass Spectrometry (UPLC-MS/MS)
10.21769/bioprotoc.2715 · 2018
Lipid extraction by methyl-tert-butyl ether for high-throughput lipidomics
10.1194/jlr.d700041-jlr200 · 2008
Bisphenol A concentrations in maternal breast milk and infant urine
10.1007/s00420-012-0834-9 · 2014
Effects of perinatal exposure to BPA and its alternatives (BPS, BPF and BPAF) on hepatic lipid and glucose homeostasis in female mice adolescent offspring
10.1016/j.chemosphere.2018.08.076 · 2018
A critical review to identify data gaps and improve risk assessment of bisphenol A alternatives for human health
10.1080/10408444.2024.2388712 · 2024
Perinatal and postnatal exposure to bisphenol a increases adipose tissue mass and serum cholesterol level in mice
10.5551/jat.e486 · 2007
Bisphenol A (BPA) and cell signaling pathways
10.1016/j.biotechadv.2017.12.002 · 2018
Bisphenol A and its analogues: from their occurrence in foodstuffs marketed in Europe to improved monitoring strategies-a review of published literature from 2018 to 2023
10.1007/s00204-024-03793-4 · 2024
Sex-Specific Modulation of Fetal Adipogenesis by Gestational Bisphenol A and Bisphenol S Exposure
10.1210/en.2017-00615 · 2017
Blood and urinary bisphenol A concentrations in children, adults, and pregnant women from china: partitioning between blood and urine and maternal and fetal cord blood
10.1021/es303808b · doi-reference
Associations of bisphenol A exposure with metabolic syndrome and its components: A systematic review and meta-analysis
10.1111/obr.13738 · doi-reference
Dihydroceramide- and ceramide-profiling provides insights into human cardiometabolic disease etiology
10.1038/s41467-022-28496-1 · doi-reference
Lipidomics Workflow for Analyzing Lipid Profiles Using Multiple Reaction Monitoring (MRM) in Liver Homogenate of Mice with Non-alcoholic Steatohepatitis (NASH)
10.21769/bioprotoc.4773 · doi-reference
Systematic Review and Meta-Analysis of Early-Life Exposure to Bisphenol A and Obesity-Related Outcomes in Rodents
10.1289/ehp1233 · doi-reference
Enhancing de novo ceramide synthesis induced by bisphenol A exposure aggravates metabolic derangement during obesity
10.1016/j.molmet.2023.101741 · doi-reference
Programming of metabolic effects in C57BL/6JxFVB mice by exposure to bisphenol A during gestation and lactation
10.1016/j.tox.2014.04.001 · doi-reference
Estrogen-related receptor γ is an in vivo receptor of bisphenol A
10.1096/fj.13-240465 · doi-reference
Prenatal Exposure to Bisphenol A Disrupts Naturally Occurring Bimodal DNA Methylation at Proximal Promoter of fggy, an Obesity-Relevant Gene Encoding a Carbohydrate Kinase, in Gonadal White Adipose Tissues of CD-1 Mice
10.1210/en.2017-00711 · doi-reference
Widely targeted quantitative lipidomics reveal lipid remodeling in adipose tissue after long term of the combined exposure to bisphenol A and fructose
10.1177/09603271241232609 · doi-reference
Anti-thyroid hormone activity of bisphenol A, tetrabromobisphenol A and tetrachlorobisphenol A in an improved reporter gene assay
10.1016/j.tiv.2009.05.004 · doi-reference
Ceramides as the molecular link between impaired lipid metabolism, saturated fatty acid intake and insulin resistance: are all saturated fatty acids to be blamed for ceramide-mediated lipotoxicity?
10.1017/s0954422424000179 · doi-reference
Environmental endocrine disruptors promote adipogenesis in the 3T3-L1 cell line through glucocorticoid receptor activation
10.1038/oby.2009.419 · doi-reference
Perinatal BPA exposure alters body weight and composition in a dose specific and sex specific manner: The addition of peripubertal exposure exacerbates adverse effects in female mice
10.1016/j.reprotox.2016.07.020 · doi-reference
Androgen and Progesterone Receptors Are Targets for Bisphenol A (BPA), 4-Methyl-2,4-bis-(P-Hydroxyphenyl)Pent-1-Ene--A Potent Metabolite of BPA, and 4-Tert-Octylphenol: A Computational Insight
10.1371/journal.pone.0138438 · doi-reference
Combined Exposure to Endocrine Disruptors, BPA and BP-3, during Pregnancy and Lactation Alters Postnatal Body Mass, Growth, White Adipose Tissue Morphology, and Adipogenic Gene Expression in a Sex-Specific Manner
10.1021/envhealth.5c00679 · doi-reference