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CD8(+) T cells mediate antiviral response in severe fever with thrombocytopenia syndrome
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CD4 T cell loss and Th2 and Th17 bias are associated with the severity of severe fever with thrombocytopenia syndrome (SFTS)
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Calcium channel blockers reduce severe fever with thrombocytopenia syndrome virus (SFTSV) related fatality
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Arginine deficiency is involved in thrombocytopenia and immunosuppression in severe fever with thrombocytopenia syndrome
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Clinical effect and antiviral mechanism of T-705 in treating severe fever with thrombocytopenia syndrome
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Clinical efficacy and safety evaluation of favipiravir in treating patients with severe fever with thrombocytopenia syndrome
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Molecular mechanism of potently neutralizing human monoclonal antibodies against severe fever with thrombocytopenia virus infection
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A rationally designed cocktail of nanobodies elicited by heterologous vaccination confers protection against SFTSV in preclinical models
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Bispecific antibodies targeting two glycoproteins on SFTSV exhibit synergistic neutralization and protection in a mouse model
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Alterations of Gut Microbiome in the Patients With Severe Fever With Thrombocytopenia Syndrome
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FXR inhibition may protect from SARS-CoV-2 infection by reducing ACE2
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The Intestinal Microbiome Restricts Alphavirus Infection and Dissemination through a Bile Acid-Type I IFN Signaling Axis
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Akkermansia muciniphila protects mice against an emerging tick-borne viral pathogen
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Taurolithocholic acid protects against viral haemorrhagic fever via inhibition of ferroptosis
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The Nonstructural Protein NSs of Severe Fever with Thrombocytopenia Syndrome Virus Causes a Cytokine Storm through the Hyperactivation of NF-kappaB
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Activation of the NLRP3 inflammasome and elevation of interleukin-1beta secretion in infection by sever fever with thrombocytopenia syndrome virus
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The NF-kappaB inhibitor, SC75741, is a novel antiviral against emerging tick-borne bandaviruses
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Host cytokine storm is associated with disease severity of severe fever with thrombocytopenia syndrome
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Correlation of cytokine level with the severity of severe fever with thrombocytopenia syndrome
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The hnRNP A2B1 is important for the replication of SFTSV and other RNA viruses
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Bunyavirus SFTSV nucleoprotein exploits tufm-mediated mitophagy to impair antiviral innate immunity
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N6-methyladenosine RNA modification promotes Severe Fever with Thrombocytopenia Syndrome Virus infection
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Severe fever with thrombocytopenia syndrome virus induces lactylation of m6A reader protein YTHDF1 to facilitate viral replication
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SFTSV induces liver ferroptosis through m6A-related ferritinophagy
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Severe Fever with Thrombocytopenia Syndrome Virus NSs Interacts with TRIM21 To Activate the p62-Keap1-Nrf2 Pathway
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SAFA initiates innate immunity against cytoplasmic RNA virus SFTSV infection
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Differential Antagonism of Human Innate Immune Responses by Tick-Borne Phlebovirus Nonstructural Proteins
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Two Conserved Amino Acids within the NSs of Severe Fever with Thrombocytopenia Syndrome Phlebovirus Are Essential for Anti-interferon Activity
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Interaction between the SFTSV envelope glycoprotein Gn and STING inhibits the formation of the STING-TBK1 complex and suppresses the NF-kappaB signaling pathway
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Virus-induced p38 MAPK activation facilitates viral infection
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SFTSV nucleoprotein mediates DNA sensor cGAS degradation to suppress cGAS-dependent antiviral responses
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The Nuclear Matrix Protein SAFA Surveils Viral RNA and Facilitates Immunity by Activating Antiviral Enhancers and Super-enhancers
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SFTSV Infection Induces BAK/BAX-Dependent Mitochondrial DNA Release to Trigger NLRP3 Inflammasome Activation
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Incoming RNA virus nucleocapsids containing a 5'-triphosphorylated genome activate RIG-I and antiviral signaling
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RIG-I-Like Receptor and Toll-Like Receptor Signaling Pathways Cause Aberrant Production of Inflammatory Cytokines/Chemokines in a Severe Fever with Thrombocytopenia Syndrome Virus Infection Mouse Model
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The pathogenesis of severe fever with thrombocytopenia syndrome virus infection in alpha/beta interferon knockout mice: insights into the pathologic mechanisms of a new viral hemorrhagic fever
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Extracellular Vesicles Mediate Receptor-Independent Transmission of Novel Tick-Borne Bunyavirus
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