Abstract
This study aims to present an integrative review and propose an interpretive matrix — grounded in the authors’ original clinical observations — addressing the intersection between Autism Spectrum Disorder (ASD), anomic aphasia, identity development, and persistent behavioral patterns, while examining the underlying biological mechanisms (gut microbiota, mitochondrial dysfunction, and atypical sensory processing) that amplify these linguistic and behavioral deficits. Through interdisciplinary neuropsychiatric-linguistic analysis, an innovative clinical hypothesis is proposed articulating language deficits (naming and concept structuring), oromotor skills, difficulties in identity formation, and the execution of complex commands. The literature review of the past five years (2021–2026) supports the need to understand "anomic aphasia" not merely as an isolated motor or verbal deficit, but as a central element in the organization of self-concept, behavioral regulation, and the persistence of obsessive patterns in ASD. Additionally, original clinical concepts are systematized: dilettantism as a self-regulation mechanism, the kinetic versus thermal energy metaphor, parental emotional anomia, growing inner void, and the refusal of instrumentalization as identity expression. The role of gut microbiota, mitochondrial dysfunction, and atypical sensory processing are discussed as underlying mechanisms that amplify linguistic and behavioral deficits. This framework offers significant clinical implications for differential assessment, self-regulation, and interdisciplinary therapeutic planning.