A noncanonical PWI domain in the N-terminal helicase-associated region of the spliceosomal Brr2 protein
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ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage
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Substrate specificities and identification of putative substrates of ATM kinase family members
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PhosphoSitePlus, 2014: mutations, PTMs and recalibrations
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The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)
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MCM8IP activates the MCM8-9 helicase to promote DNA synthesis and homologous recombination upon DNA damage
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Control of homologous recombination by the HROB-MCM8-MCM9 pathway
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XRCC3 controls the fidelity of homologous recombination: roles for XRCC3 in late stages of recombination
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Differential regulation of short- and long-tract gene conversion between sister chromatids by Rad51C
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XRCC2 and XRCC3 regulate the balance between short- and long-tract gene conversions between sister chromatids
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The human HELQ helicase and XRN2 exoribonuclease cooperate in R-loop resolution
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The archaeal ATPase PINA interacts with the helicase Hjm via its carboxyl terminal KH domain remodeling and processing replication fork and Holliday junction
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Hjm/Hel308A DNA helicase from Sulfolobus tokodaii promotes replication fork regression and interacts with Hjc endonuclease in vitro
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Communication between DNA polymerases and replication protein A within the archaeal replisome
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Role of RadA and DNA polymerases in recombination-associated DNA synthesis in hyperthermophilic archaea
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Mitotic DNA synthesis in untransformed human cells preserves common fragile site stability via a FANCD2-driven mechanism that requires HELQ
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Mitotic DNA synthesis in response to replication stress requires the sequential action of DNA polymerases zeta and delta in human cells
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Interaction of human HelQ with DNA polymerase delta halts DNA synthesis and stimulates DNA single-strand annealing
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Break-induced replication is a source of mutation clusters underlying kataegis
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DNA polymerase delta synthesizes both strands during break-induced replication
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Migrating bubble during break-induced replication drives conservative DNA synthesis
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Break-induced replication is highly inaccurate
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Hallmarks of cancer: the next generation
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HELQ upregulates PARP1 to drive platinum resistance and predict therapeutic response in ovarian cancer
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DNA repair protein HELQ and XAB2 as chemoresponse and prognosis biomarkers in ascites tumor cells of high-grade serous ovarian cancer
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Helicase POLQ-like (HELQ) as a novel indicator of platinum-based chemoresistance for epithelial ovarian cancer
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Defining a cancer dependency map
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HELQ is a dual-function DSB repair enzyme modulated by RPA and RAD51
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Division of labor by the HELQ, BLM, and FANCM helicases during homologous recombination repair in Drosophila melanogaster
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Overlapping mechanisms promote postsynaptic RAD-51 filament disassembly during meiotic double-strand break repair
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Helicase Q promotes homology-driven DNA double-strand break repair and prevents tandem duplications
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Caenorhabditis elegans POLQ-1 and HEL-308 function in two distinct DNA interstrand cross-link repair pathways
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Complete sequencing and characterization of 21,243 full-length human cDNAs
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Human HEL308 localizes to damaged replication forks and unwinds lagging strand structures
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Helq acts in parallel to Fancc to suppress replication-associated genome instability
10.1093/nar/gkt676 · doi-reference
Human DNA helicase HELQ participates in DNA interstrand crosslink tolerance with ATR and RAD51 paralogs
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HELQ promotes RAD51 paralogue-dependent repair to avert germ cell loss and tumorigenesis
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Archaeal Hel308 suppresses recombination through a catalytic switch that controls DNA annealing
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