Abstract
Thomas A. Milne
Abstract
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10.1128/mcb.24.13.5639-5649.2004 · 2004
The menin tumor suppressor protein is an essential oncogenic cofactor for MLL-associated leukemogenesis
10.1016/j.cell.2005.09.025 · 2005
Targeting chromatin regulators inhibits leukemogenic gene expression in NPM1 mutant leukemia
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MEN1 mutations mediate clinical resistance to menin inhibition
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10.1016/s0092-8674(00)80967-8 · 1999
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