GEPIA: a web server for cancer and normal gene expression profiling and interactive analyses
10.1093/nar/gkx247 · doi-reference
Normal cells, but not cancer cells, survive severe Plk1 depletion
10.1128/mcb.26.6.2093-2108.2006 · doi-reference
Three-dimensional (3D) tumor spheroid invasion assay
10.3791/52686 · doi-reference
Cobimetinib combined with vemurafenib in advanced BRAF(V600)-mutant melanoma (coBRIM): updated efficacy results from a randomised, double-blind, phase 3 trial
10.1016/s1470-2045(16)30122-x · doi-reference
Survival of patients with advanced metastatic melanoma: the impact of novel therapies-update 2017
10.1016/j.ejca.2017.06.028 · doi-reference
Clinical development of BRAF plus MEK inhibitor combinations
10.1016/j.trecan.2020.05.009 · doi-reference
Phosphorylation of BACH1 switches its function from transcription factor to mitotic chromosome regulator and promotes its interaction with HMMR
10.1042/bcj20170520 · doi-reference
Antioxidant-induced phosphorylation of tyrosine 486 leads to rapid nuclear export of Bach1 that allows Nrf2 to bind to the antioxidant response element and activate defensive gene expression
10.1074/jbc.m109.040022 · doi-reference
Nrf2 activation promotes lung cancer metastasis by inhibiting the degradation of Bach1
10.1016/j.cell.2019.06.003 · doi-reference
Heme mediates derepression of Maf recognition element through direct binding to transcription repressor Bach1
10.1093/emboj/20.11.2835 · doi-reference
The transcription factor Bach1 suppresses the developmental angiogenesis of zebrafish
10.1155/2017/2143875 · doi-reference
Bach1 inhibits oxidative stress-induced cellular senescence by impeding p53 function on chromatin
10.1038/nsmb.1516 · doi-reference
Polo-like kinase 1 coordinates biosynthesis during cell cycle progression by directly activating pentose phosphate pathway
10.1038/s41467-017-01647-5 · doi-reference
Targeted knockdown of polo-like kinase 1 alters metabolic regulation in melanoma
10.1016/j.canlet.2017.02.013 · doi-reference
Targeting mitochondrial biogenesis to overcome drug resistance to MAPK inhibitors
10.1172/jci82661 · doi-reference
Antioxidants accelerate lung cancer progression in mice
10.1126/scitranslmed.3007653 · doi-reference
Glutathione and thioredoxin antioxidant pathways synergize to drive cancer initiation and progression
10.1016/j.ccell.2014.11.019 · doi-reference
